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When cells become resistant to insulin, the receptors on their surfaces designed to respond to insulin have begun to malfunction. It simply means that the receptors require more insulin to make them work properly in removing sugar from the blood. Whereas before they needed just a touch to lower it, now they need a continuous supply of excess insulin to keep blood sugar within normal range.

As time goes by, blood sugar rises higher and stays up longer after the carbohydrate meal despite the enormous amount of insulin mustered to lower it. Bear in mind that were your doctor to check blood sugar during this stage of developing insulin resistance, your blood sugar would be perfectly normal. The major silent change taking place is the ever-growing quantity of insulin needed to keep it that way.

The liver becomes resistant first, then the muscle tissue, then the fat. What is the effect of insulin on the liver? It is to suppress the production of sugar by the liver.

In the morning you will find that your sugar level is more from what your liver has produced over night rather than what you have ingested. If the liver is responding to insulin correctly it would not make much sugar at all during the night. If the liver is not responding right then there will be no way to control the amount of sugar it makes.

After your liver stops responding to insulin your muscles will be next. The muscles are designed to burn sugar that is made by your liver. If they are not responding to insulin the will not know that they need to burn the sugar. If you haven’t noticed there is a vicious chain going on, your liver is making too much sugar and your muscles can’t burn it off. So now what is affected?

Next are the fat cells that are affected by poor insulin. The fat cells are what stores the fat. They take sugar and store it as fat. Therefore, until your fat cells become resistant to insulin you are likely to gain more weight. You may gain a little or you may gain a lot, but what matters is that you are gaining weight and you need to know why.

So for a while your fat cells retain their sensitivity. What is the action of insulin on your fat cells? To store that fat. It takes sugar and it stores it as fat. So until your fat cells become resistant you get fat, and that is what you see. As people become more and more insulin resistant, they get fat and their weight goes up.

But eventually they plateau. They might plateau at three hundred pounds, two hundred and twenty pounds, one hundred and fifty pounds, but they will eventually plateau as the fat cells protect themselves and become insulin resistant.

As all these major tissues, this massive body becomes resistant, your liver, muscles and fat, your pancreas is putting out more insulin to compensate, so you are hyperinsulinemic [having an abnormally high level of insulin in the blood] and you’ve got insulin floating around all the time.

Insulin floating around in the blood causes a plaque build up. Insulin causes the blood to clot too readily. Insulin causes cells that accumulate fatty deposits. Every step of the way, insulin’s got its fingers in it and is causing cardiovascular disease. It fills it with plaque, it constricts the arteries, it increases platelet adhesiveness and ability of the blood to coagulate [clot]. Any known cause of cardiovascular disease, insulin is a part of.

If you want to know if insulin sensitivity can be restored to its original state, well, perhaps not to its original state, but you can restore it to the state of about a ten year old.

You can increase sensitivity by diet and a lot of supplements.